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Oxidative stress alters base excision repair pathway and increases apoptotic response in apurinic/apyrimidinic endonuclease 1/redox factor-1 haploinsufficient mice

Unnikrishnan, A, Raffoul, JJ, Patel, HV, Prychitko, TM, Anyangwe, N, Meira, LB, Friedberg, EC, Cabelof, DC and Heydari, AR (2009) Oxidative stress alters base excision repair pathway and increases apoptotic response in apurinic/apyrimidinic endonuclease 1/redox factor-1 haploinsufficient mice FREE RADICAL BIOLOGY AND MEDICINE, 46 (11). pp. 1488-1499.

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Item Type: Article
Authors :
NameEmailORCID
Unnikrishnan, AUNSPECIFIEDUNSPECIFIED
Raffoul, JJUNSPECIFIEDUNSPECIFIED
Patel, HVUNSPECIFIEDUNSPECIFIED
Prychitko, TMUNSPECIFIEDUNSPECIFIED
Anyangwe, NUNSPECIFIEDUNSPECIFIED
Meira, LBl.meira@surrey.ac.ukUNSPECIFIED
Friedberg, ECUNSPECIFIEDUNSPECIFIED
Cabelof, DCUNSPECIFIEDUNSPECIFIED
Heydari, ARUNSPECIFIEDUNSPECIFIED
Date : 1 June 2009
Identification Number : https://doi.org/10.1016/j.freeradbiomed.2009.02.021
Uncontrolled Keywords : Science & Technology, Life Sciences & Biomedicine, Biochemistry & Molecular Biology, Endocrinology & Metabolism, BIOCHEMISTRY & MOLECULAR BIOLOGY, ENDOCRINOLOGY & METABOLISM, Apurinic/apyrimidinic endonuclease 1/redox factor-1, Redox activity, Base excision repair, Oxidative DNA damage, NF-kappa B, Apoptosis, Liver, Free radicals, NF-KAPPA-B, POLYMERASE-BETA HAPLOINSUFFICIENCY, STRANDED DEOXYRIBONUCLEIC-ACID, URACIL-DNA GLYCOSYLASE, HUMAN-CELLS, BINDING ACTIVITY, DAMAGING AGENTS, APURINIC SITES, NITRIC-OXIDE, ACTIVATION
Related URLs :
Depositing User : Symplectic Elements
Date Deposited : 17 May 2017 08:58
Last Modified : 17 May 2017 14:38
URI: http://epubs.surrey.ac.uk/id/eprint/821713

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