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New immunoaffinity-LC-MS/MS methodology reveals that Aag null mice are deficient in their ability to clear 1,N-6-etheno-deoxyadenosine DNA lesions from lung and liver in vivo

Ham, AJL, Engelward, BP, Koc, H, Sangaiah, R, Meira, LB, Samson, LD and Swenberg, JA (2004) New immunoaffinity-LC-MS/MS methodology reveals that Aag null mice are deficient in their ability to clear 1,N-6-etheno-deoxyadenosine DNA lesions from lung and liver in vivo DNA REPAIR, 3 (3). pp. 257-265.

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Item Type: Article
Authors :
NameEmailORCID
Ham, AJLUNSPECIFIEDUNSPECIFIED
Engelward, BPUNSPECIFIEDUNSPECIFIED
Koc, HUNSPECIFIEDUNSPECIFIED
Sangaiah, RUNSPECIFIEDUNSPECIFIED
Meira, LBl.meira@surrey.ac.ukUNSPECIFIED
Samson, LDUNSPECIFIEDUNSPECIFIED
Swenberg, JAUNSPECIFIEDUNSPECIFIED
Date : 4 March 2004
Identification Number : https://doi.org/10.1016/j.dnarep.2003.11.003
Uncontrolled Keywords : Science & Technology, Life Sciences & Biomedicine, Genetics & Heredity, Toxicology, GENETICS & HEREDITY, TOXICOLOGY, immunoaffinity-LC-MS/MS methodology, 1,N-6-ethenodeoxyadenosine, lung, liver, alkyladenine DNA glycosylase, mice, NUCLEOTIDE EXCISION-REPAIR, OXIDATIVE STRESS MARKERS, ETHYL CARBAMATE URETHANE, VINYL-CHLORIDE, LIPID-PEROXIDATION, ESCHERICHIA-COLI, POTENTIAL ROLE, MASS-SPECTROMETRY, FORMING CHEMICALS, MOUSE CELLS
Related URLs :
Depositing User : Symplectic Elements
Date Deposited : 17 May 2017 08:54
Last Modified : 17 May 2017 14:38
URI: http://epubs.surrey.ac.uk/id/eprint/821417

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